Reinstatement of a stored representation from an incomplete or degraded cue. Recurrent autoassociative memory provides one mechanism; its success depends on the cue entering the correct basin of attraction. Partial-cue deficits after CA3-targeted NMDA receptor deletion support a hippocampal role without identifying a unique weight-update rule.[ref]
Requirement for hippocampal CA3 NMDA receptors in associative memory recall by Kazu Nakazawa and others. Abstract; pp. 211–218, adult CA3-restricted deletion and full-cue versus partial-cue recall
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