Current STI treatment guidance: ceftriaxone-based treatment, microbiological testing and management of partners.
London: John Churchill and Sons, 1864, 34 pages. Full title continues “being an essay on trichinosis, or flesh-worm disease, its prevention and cure”. Original scan: upload.wikimedia.org/wikipedia/commons/0/0d/On_poisoning_by_diseased_pork_-_being_an_essay_on_trichinosis%2C_or_flesh-worm_disease%2C_its_prevention_and_cure_%28IA_b22267372%29.pdf. The opening discussion connects pork, trichinae and microscopic examination; the historical remedies are not present treatment advice.
Present institutional account of infection through animal products and contaminated meat.
Infectious agent, faecal–oral transmission, clinical presentation, blood culture and complications.
Current topical and systemic antifungal treatment, distinct from nineteenth-century remedies.
Clinical examination and microscopy of samples from mouth or throat lesions.
Mouth and throat candidiasis: host conditions and medication-related risks.
Symptoms in adults and infants, treatment and feeding-equipment hygiene.
Chapter 7, clinical features and the adherent pseudomembrane.
Clinical features, including respiratory prodrome and evolution of the rash.
Early and late latent regimens, disease of unknown duration, pregnancy and follow-up.
Recommended adult regimen and special considerations; clinical and serological follow-up.
3 September 1864, p. 263. Historical late manifestations and treatment with iodide, sometimes combined with mercury.
Contemporary article, pp. 17–23, comparing mercurial and non-mercurial treatment and describing uncertainty and adverse effects.
NG84, self-care, selective antibiotics, severe illness and referral.
Staging, diagnosis and follow-up; linked stage-specific treatment recommendations.
Diagnosis, antibiotic susceptibility and intestinal complications.
Clinical features, complications, diagnostic limitations and doxycycline treatment.
Two-page clinical diagnostic poster: major and minor criteria, ordinary smallpox evolution and distribution, and comparison with chickenpox. It is a modern diagnostic resource, not a record of an 1865 patient.
Ordinary, modified, flat and haemorrhagic types; modified disease after vaccination.
Major and minor diagnostic criteria; differential diagnosis; laboratory confirmation.
Overview: increased risk of progression from tuberculosis infection to active disease.
Histopathology: classical congestion, red hepatization, grey hepatization and resolution; the untreated sequence is a model, not a universal timetable.
Fact sheet, 30 January 2025, transmission and life cycle: infected copepods swallowed in water. This explanatory life cycle is not attributed to the 1865 candidate.
Original memoir by Louis Pasteur, Annales scientifiques de l’École Normale Supérieure, first series, volume 1, pages 113–158. DOI: doi.org/10.24033/asens.4. The numbered sections discuss acetification, the surface growth and experimental conditions.
Guidance on organic contamination, germicide consumption and contact duration.
Current institutional guidance from the Centers for Disease Control and Prevention. Cited sections: Bacteriologic examination, nucleic acid amplification and culture. Used only for the modern distinction between chemical composition and infectious diagnosis, not as an 1866 laboratory procedure.
Original analyses in Robert Mortimer Glover's On the Pathology and Treatment of Scrofula, reproduced as quoted primary passages in the contemporary article Dr. Glover on Scrofula. Inspected reproduction: printed pp. 516–517, explicitly locating the first quoted original analysis at book p. 73. It supports solvent fractionation, protein-rich residue, mineral salts and variation between lesions. The entire original book was not inspected; therapeutic and etiological beliefs in the surrounding review are not endorsed.
Contemporary book by Félix Archimède Pouchet, published by J. B. Baillière in Paris, defending spontaneous generation.
Original memoir by Louis Pasteur; atmospheric particles, boiled liquids, curved-neck flasks and the spontaneous-generation controversy.
Original memoir by Louis Pasteur, communicated in 1857 and printed in 1858; available here in the collected works, volume II, pp. 3–13. §II gives inoculation and growth experiments and contrasts the rival chemical theory.
Purulent skin and soft tissue infections; furuncles and carbuncles; cultures, incision and drainage and adjunctive antibiotics.
Immune globulin; control of spasms; airway, wound and antimicrobial management; vaccination during recovery.
Indian Medical Gazette 1(11), 1 November 1866, pp. 336–337. Report signed with initials A. S. J.; no full identity is inferred. The historical heading uses Anœsthesia. Individual interventions and outcome do not establish comparative efficacy.

Clinical Guidelines for Post-Streptococcal Glomerulonephritis by CDC

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www.cdc.gov/group-a-strep/hcp/clinical-guidance/post-streptococcal-glomerulonephritis.html
Clinical features, treatment and prevention; evidence is insufficient that antimicrobial therapy prevents PSGN.
Testing, treatment, complications and prevention.
B 39, issue 3.2 (October 2025). Microscopy and specimen types; direct preparations, culture and their limitations.
Histology: Malassezia yeasts and short hyphae in the stratum corneum.
Clinical features and diagnosis, hair involvement and microscopy with culture.
Histology; Special stains; Differential diagnosis, including eczema and psoriasis.
Introduction and Etiology: scutula, scarring and fungal nomenclature.
Hair-shaft invasion, endothrix versus ectothrix, clinical variants and mycological investigation.
Contemporary review published April 1864. Discussion of the conflicting fungal classifications and historical assertions about tinea decalvans. These assertions are not adopted as modern conclusions.
Intestinal tuberculosis: ulcerative and ulcero-hypertrophic patterns, ileocaecal disease, caseating granulomas and complications.
Salmonella, typhoid histology: lymphoid follicles, macrophages and deep ulcers.
Histopathology: macrophage-rich nodules, lymphoid hyperplasia and intestinal ulceration. Current indexed entry displays update September 2026.
Histopathological diagnosis: pulmonary granulomas, caseous necrosis, epithelioid cells and giant cells; limits of morphology alone.
Risk factors; How it spreads: exposure, progression and airborne particles reaching alveoli.
Original Communications, p. 913: contemporary discussion of inadequate diet and crowded, ill-ventilated accommodation; not a modern dietary recommendation.
“Natural ventilation in the home” explains that top window openings allow incoming fresh air to mix and warm, reducing cold draughts. This present-day guidance supports the physical mixing explanation; it is not cited as an 1866 publication.
Official explanation of airborne transmission and poorly ventilated indoor exposure.
“Causes and spread”: human carriers, excreta, contaminated food and water.
Part III, British Medical Journal, 30 November 1861, p.575. The opening propositions place the specific infectious material in intestinal discharges and reject spontaneous production from decay.
Glasgow Medical Journal, October 1867, pp. 201–211. James Burn Russell distinguishes supposed stimulation from narcosis and criticizes fixed high-dose schedules. Cited for historical practice and debate, not modern efficacy.
Selection of regimen according to susceptibility, comorbid conditions and interactions.
NG253; high-risk management, intravenous fluid resuscitation and reassessment.
Scalp treatment and adjunctive shampoo to reduce household reinfection.
Treatment by anatomical site; scalp infection and caution about topical steroids.
Diagnosis, treatment and prevention.
Diagnostic assessment; observation and local treatment of limited disease.
Microscopy, examination lamp, treatment and delayed pigment recovery.
Clinical description and Patient advice.
Chapter 7, management of chorea, especially Table 7.6.
Clinical manifestations; carditis and chorea; antibiotic treatment and recurrence prevention.
Chicago Medical Examiner 3, pp. 553–557, by William Brinton. The catalogue spells “Tuburcular”; the article heading spells “Tubercular”. Period criticism of competing remedies and therapeutic inference.
Edinburgh Medical Journal 13, pp. 140–157, by Thomas John Maclagan. Table V and discussion, pp. 154–157: recorded beverage quantities are observational average maxima, not standard doses.
Edinburgh Medical Journal 12, pp. 1–29. Period primary clinical report by Alexander Smith (military physician); describes practice and selection of cases, not modern efficacy evidence.
Etiology; Symptoms and Signs; Diagnosis, including distinction between airway colonization and infection. Page displays May 2026 update.
Infant symptoms and signs of breathing problems. Page dated March 24, 2022.
Bronchopneumonia: Pathologic Features, airway-centred exudation, patchy involvement and coalescence.
Introduction; Histopathology, including lobar pneumonia and bronchopneumonia.
Official parasite identification and life-cycle resource: www.cdc.gov/dpdx/
Cited passages: Causal Agents; Life Cycle.
Cited passages: Causal Agents; Life Cycle.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Cited sections: Causal Agents or Causal Agent; Life Cycle; Geographic Distribution; Laboratory Diagnosis when applicable.
Contemporary monograph by Thomas Spencer Cobbold, published by Groombridge in London. Includes separate treatments of Trematoda, Nematoda, Acanthocephala and Cestoda.
Cloning and heterologous pathway reconstruction identify core phenazine loci and modifying enzymes.
Binding-interface mutants with retained catalytic activity separate PqsE–RhlR regulation from PqsE catalysis.
Phenazine-mutant and supplementation experiments link pyocyanin to intracellular redox balance under oxygen limitation.
Loss-of-function and osmotic-downshock experiments demonstrate emergency channel protection from lysis.
Live fluorescence measurements link circumferential MreB-associated movement to peptidoglycan synthesis.
Outer-membrane perturbation, ferricyanide reduction and supernatant substitution tests identify a barrier to cyanobacterial electron export.
Oriented MtrCAB proteoliposomes transfer electrons from an internal donor pool to external iron oxides.
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