An energetic neutron can collide with a lattice atom and create a primary knock-on atom. That recoil transfers energy to other atoms in a displacement cascade. Some atoms leave their normal lattice sites, creating Frenkel pairs: a vacancy and an extra atom at an interstitial position.
Many nearby defects recombine during the cascade and its thermal relaxation. The surviving point defects are therefore fewer than a simple count of all displacement events. Some defects are already clustered within the cascade; others migrate and aggregate later.
A collection of vacancies can collapse into a platelet bounded by a vacancy-type dislocation loop. An aggregation of self-interstitial atoms can create an extra atomic platelet bounded by an interstitial-type dislocation loop. The boundary is a line defect because a circuit around it has a nonzero Burgers vector. The allowed Burgers vectors and habit planes depend on the material's crystal structure and fault energies.
Loops can grow by absorbing mobile point defects, shrink by emitting or absorbing the opposite defect type, and interact with existing dislocations. Some remain faulted, while suitable loops can unfault or become part of a larger dislocation network. Repeated cascades and subsequent defect transport therefore create a population of extended defects even in material with a low initial dislocation density.
This describes the main displacement-damage mechanism. Ionizing energy deposited in a material does not automatically create the same defect population as a neutron recoil cascade; recoil spectrum, displacement thresholds and material response matter.
Figure 1.
Original schematic of a displaced atom, a Frenkel pair, and the competing pathways to recombination, sinks, loops and voids
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