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Coauthor of research on multisite control of Cdc25. The study connected phosphorylation at several sites with the timing and sharpness of entry into and exit from mitosis.^[ref]

Coauthor of research on multisite control of Cdc25. The study connected phosphorylation at several sites with the timing and sharpness of entry into and exit from mitosis.^[ref]

Coauthor of research on multisite control of Cdc25. The study connected phosphorylation at several sites with the timing and sharpness of entry into and exit from mitosis.^[ref]

Coauthor of research on hysteresis and bistability in Cdc2 activation. The work connected feedback in the cell cycle network with abrupt switches and different activation and deactivation thresholds.^[ref]

Coauthor of research on hysteresis and bistability in Cdc2 activation. The work connected feedback in the cell cycle network with abrupt switches and different activation and deactivation thresholds.^[ref]

Coauthor of research on hysteresis and bistability in Cdc2 activation. The work connected feedback in the cell cycle network with abrupt switches and different activation and deactivation thresholds.^[ref]

Coauthor of research on the precision and speed of adaptation in bacterial chemotaxis. The work connected recovery of a signaling output with its time course, distinguishing an endpoint response from the mechanism that produces it.^[ref]

Coauthor of research on the precision and speed of adaptation in bacterial chemotaxis. The work connected recovery of a signaling output with its time course, distinguishing an endpoint response from the mechanism that produces it.^[ref]

Coauthor of research on the precision and speed of adaptation in bacterial chemotaxis. The work connected recovery of a signaling output with its time course, distinguishing an endpoint response from the mechanism that produces it.^[ref]

Coauthor of research on the precision and speed of adaptation in bacterial chemotaxis. The work connected recovery of a signaling output with its time course, distinguishing an endpoint response from the mechanism that produces it.^[ref]

Coauthor of research on receptor sensitivity in bacterial chemotaxis. The study connected changes in extracellular chemical input with amplified intracellular signaling, making receptor interactions relevant to behavioral sensitivity.^[ref]

Coauthor of research on receptor sensitivity in bacterial chemotaxis. The study connected changes in extracellular chemical input with amplified intracellular signaling, making receptor interactions relevant to behavioral sensitivity.^[ref]