Exact division refers to the process of dividing one number by another where the result is a whole number without any remainder.
Ernie Tate is not widely recognized as a notable figure or brand as of my last knowledge update in October 2023.
Sodium channels are integral membrane proteins that facilitate the movement of sodium ions (Na⁺) across the cell membrane. They play a crucial role in a variety of physiological processes, including the generation and propagation of action potentials in neurons and muscle cells.
The alpha-4 beta-2 nicotinic acetylcholine receptor (α4β2 nAChR) is a type of receptor in the nervous system that responds to the neurotransmitter acetylcholine as well as nicotine, a substance found in tobacco. It is a subtype of the nicotinic acetylcholine receptor family, which are ligand-gated ion channels playing crucial roles in neurotransmission.
CACNA1G is a gene that encodes a subunit of a voltage-gated calcium channel. Specifically, it is part of the family of calcium channel genes that contribute to the formation of L-type calcium channels, which are essential for a variety of physiological processes, including muscle contraction, neurotransmitter release, and neuronal excitability. The protein produced by the CACNA1G gene is particularly involved in the regulation of calcium ions flowing into cells in response to changes in membrane potential.
CACNA2D1 is a gene that encodes a subunit of a voltage-gated calcium channel. Specifically, it encodes the alpha-2/delta-1 subunit of the calcium channel complex. Voltage-gated calcium channels play a crucial role in various physiological processes, including muscle contraction, neurotransmitter release, and gene expression.
CLCN5 is a gene that encodes a chloride channel protein, which is a member of the CLC (chloride channel) family of proteins. This family plays an essential role in various physiological processes, including the regulation of ion balance, electrical excitability of cells, and the maintenance of acid-base homeostasis. The CLCN5 protein specifically is primarily expressed in the kidneys, particularly in the renal proximal tubule cells, where it is involved in the reabsorption of chloride ions.
CLIC6, or Chloride Intracellular Channel protein 6, is a protein that belongs to the CLIC (Chloride Intracellular Channel) family of proteins. These proteins are characterized by their ability to form ion channels that are selective for chloride ions, which play important roles in various cellular processes, including maintaining the cell's electrochemical gradient, regulating cell volume, and signaling. CLIC6 is involved in several physiological and pathological processes.
KCNK5, also known as "Potassium Channel, Two-Pore Domain, Subfamily K, Member 5," is a gene that encodes a protein involved in the formation of a specific type of potassium ion channel. These channels are integral in various physiological processes, including setting the resting membrane potential of cells, regulating excitability, and contributing to the overall homeostasis of potassium ions within cells.
Cav1.4 refers to a specific type of voltage-gated calcium channel, which is part of the Cav1 family of channels. These channels are crucial for the influx of calcium ions (Ca²⁺) into cells in response to membrane depolarization. Cav1.4 is primarily found in the retina, particularly in photoreceptor cells (such as rods and cones) and some bipolar cells. Key features of Cav1.
GABRQ is a term that could refer to several contexts, but it is not widely recognized as a specific acronym or term in mainstream use as of my last knowledge update in October 2023. It may refer to a product, service, or concept within a specific field or emerging technology that is less known.
KCNA10, or Potassium Voltage-Gated Channel Subfamily A Member 10, is a protein that in humans is encoded by the KCNA10 gene. This protein is a member of the voltage-gated potassium channel family, which plays critical roles in the electrical excitability of cells, including those in the heart and nervous system.
KCNG4, or "Potassium Voltage-Gated Channel Subfamily G Member 4," is a gene that encodes a protein belonging to the family of voltage-gated potassium channels. These channels are essential for the regulation of electrical signaling in tissues, particularly in the nervous system and muscle tissues. KCNG4 is specifically known for its role in the transmission of electrical impulses across cell membranes.
KCNAB3 refers to a gene that encodes a protein called potassium voltage-gated channel subfamily A member 3. This protein is a part of the larger family of potassium channels, which are critical for various physiological processes, including the regulation of cardiac, neuronal, and muscle excitability. The KCNAB3 protein functions as an auxiliary subunit that can influence the properties of voltage-gated potassium channels, such as their conductance, kinetics, and membrane localization.
KCNK6, also known as TWIK-related K+ channel 6, is a gene that encodes a protein belonging to the two-pore domain potassium channel (K2P) family. These channels are known for their role in maintaining the resting membrane potential and regulating the excitability of neurons and other cells by allowing potassium ions (K+) to flow across the cell membrane. KCNK6 is characterized by its ability to contribute to the background potassium conductance in various tissues.
KCNE4 is a gene that encodes a protein belonging to the KCNE family of potassium channel accessory subunits. These proteins are known to modulate the function of voltage-gated potassium channels, influencing their gating properties, conductance, and localization. Specifically, KCNE4 is associated with the modulation of ion flow and plays a role in various physiological processes, including the regulation of cardiac rhythm and other electrical activities in cells.

Pinned article: Introduction to the OurBigBook Project

Welcome to the OurBigBook Project! Our goal is to create the perfect publishing platform for STEM subjects, and get university-level students to write the best free STEM tutorials ever.
Everyone is welcome to create an account and play with the site: ourbigbook.com/go/register. We belive that students themselves can write amazing tutorials, but teachers are welcome too. You can write about anything you want, it doesn't have to be STEM or even educational. Silly test content is very welcome and you won't be penalized in any way. Just keep it legal!
We have two killer features:
  1. topics: topics group articles by different users with the same title, e.g. here is the topic for the "Fundamental Theorem of Calculus" ourbigbook.com/go/topic/fundamental-theorem-of-calculus
    Articles of different users are sorted by upvote within each article page. This feature is a bit like:
    • a Wikipedia where each user can have their own version of each article
    • a Q&A website like Stack Overflow, where multiple people can give their views on a given topic, and the best ones are sorted by upvote. Except you don't need to wait for someone to ask first, and any topic goes, no matter how narrow or broad
    This feature makes it possible for readers to find better explanations of any topic created by other writers. And it allows writers to create an explanation in a place that readers might actually find it.
    Figure 1.
    Screenshot of the "Derivative" topic page
    . View it live at: ourbigbook.com/go/topic/derivative
  2. local editing: you can store all your personal knowledge base content locally in a plaintext markup format that can be edited locally and published either:
    This way you can be sure that even if OurBigBook.com were to go down one day (which we have no plans to do as it is quite cheap to host!), your content will still be perfectly readable as a static site.
    Figure 2.
    You can publish local OurBigBook lightweight markup files to either https://OurBigBook.com or as a static website
    .
    Figure 3.
    Visual Studio Code extension installation
    .
    Figure 4.
    Visual Studio Code extension tree navigation
    .
    Figure 5.
    Web editor
    . You can also edit articles on the Web editor without installing anything locally.
    Video 3.
    Edit locally and publish demo
    . Source. This shows editing OurBigBook Markup and publishing it using the Visual Studio Code extension.
    Video 4.
    OurBigBook Visual Studio Code extension editing and navigation demo
    . Source.
  3. https://raw.githubusercontent.com/ourbigbook/ourbigbook-media/master/feature/x/hilbert-space-arrow.png
  4. Infinitely deep tables of contents:
    Figure 6.
    Dynamic article tree with infinitely deep table of contents
    .
    Descendant pages can also show up as toplevel e.g.: ourbigbook.com/cirosantilli/chordate-subclade
All our software is open source and hosted at: github.com/ourbigbook/ourbigbook
Further documentation can be found at: docs.ourbigbook.com
Feel free to reach our to us for any help or suggestions: docs.ourbigbook.com/#contact